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This finding remained unchanged in all subgroup analyses (Table 3(Tab

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In line with reports indicating that PPAR may act on hypothalamic neurocircuitries 5,6 , we identified a series of new PPAR protein targets in the hypothalamus that are acutely upregulated by tesaglitazar and by GLP-1RA/tesaglitazar, but not by treatment with the GLP-1RA

PPAR agonists have also been shown to not be required to increase the number of PPAR-binding sites, suggesting that chromatin accessibility at PPAR motifs in SC- cells could be maintained and poised for activation in the absence of an agonist 57 , consistent with our observations above (Fig

Oocytes were first fixed in 4% paraformaldehyde and permeabilized using Triton X-100, followed by blocking in 1% BSA

In the HR (hydrophobic region) mutant (319324), a hydrophobic region -actin region that may mediate proteinprotein interactions, was deleted 15