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These findings suggest that, beyond the substrate-energy dimension, sulfur-containing amino acid-related metabolites can also regulate macrophage functional states in chronic inflammatory environments by affecting processes such as oxidative stress, protein modification, and mitochondrial homeostasis

NLRP3/cryopyrin is necessary for interleukin-1 (IL-1) release in response to hyaluronan, an endogenous trigger of inflammation in response to injury

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