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At the same time, Trp depletion itself can activate amino acid stress pathways, such as general control nonderepressible 2 (GCN2)-related responses, and suppress pro-inflammatory reactions, thereby counterbalancing M1-like programs (54)

Lowe35 states that over 50% of patients with FMS have laboratory test results consistent with either primary or central hypothyroidism, and his textbook provides a comprehensive treatise on the subject of hypothyroidism and FMS