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Furthermore, GPX4 deficient mice are associated with increased interstitial oedema and tubular cell death, suggesting a critical role for GPX4 in mediating renal tubular injury (132)

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Pharmacology Biochemistry and Behavior 92: 465-468

Research has demonstrated that KPV inhibits NF-kB nuclear translocation, a master regulator of inflammatory gene expression, and exhibits direct antimicrobial activity against several bacterial strains relevant to wound infection models