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Hyperglycaemia differentially affects energy metabolism across kidney cell types, for example impeding glycolysis in glomerular epithelial cells (where glucose is the preferred energy substrate) while enhancing glycolysis in proximal tubular epithelial cells (which typically rely on fatty acid oxidation) [11]

Treatment with the QV formulation also did not normalize the serum levels of albumin, a protein produced in the liver

Effects of empagliflozin on erythropoiesis in heart failure: data from the Empire HF trial

Ducsay CA, Myers DA: eNOS activation and NO function: differential control of steroidogenesis by nitric oxide and its adaptation with hypoxia

Recent evidence has shown that oxidative stress plays a crucial role in the pathogenesis of neuralgias

That bypass is a double-edged finding