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The expression of CD3D (T cells) and MS4A1 (B cells) was low in the control group, whereas the distribution of CD38 (plasma cella) was consistent with the results of HE staining ( Figures 6O, P , 2B )

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Similar content being viewed by others Introduction Loss of cardiomyocytes by apoptotic and/or necrotic cell death contributes to the pathogenesis of multiple forms of heart disease such as ischemia/reperfusion injury (I/R), myocarditis, cardiomyopathy, drug induced cardiotoxicity, and heart failure of diverse etiologies 1, 2