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Its probable mechanism includes activation of renal AGE-RAGE signaling pathways, decreasing levels of monocyte chemotactic protein-1 (MCP-1) and PKC-A, which consequently reduces the production of slit diaphragm proteins such as nephrin and podocin, thereby mitigating the impact of AGE-mediated DN pathogenesis (Muthenna et al., 2014)

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About this article Once-Weekly Semaglutide in Adults with Overweight or Obesity The New England Journal of Medicine (NEJM) (Mar 2021) Clinical Recommendations for the Management of Adverse Events Associated with Glucagon-Like Peptide-1 Receptor Agonists Journal of Clinical Medicine (Dec 2022) Notes:Article published

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