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As a result of decrease GSH concentration which affect the intracellular redox state will which shift more oxidants cause hepatocellular injury and release of liver enzymes (Figure 2), which could require fast reduction of oxidized cysteines allowing replenish the substrate for GSH
10.1523/JNEUROSCI.3516-16.2017 J Neurosci
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doi: 10.3390/jcm10132762 9 EppigJJ
Summary Keywords schizophrenia, first-episode psychosis, neuroleptic-nave, oxidative stress, purine catabolism, monoamine neurotransmitters Citation Yao JK, Dougherty GG, Reddy RD, Matson WR, Kaddurah-Daouk R and Keshavan MS (2013) Associations between purine metabolites and monoamine neurotransmitters in first-episode psychosis
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