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However, related studies show that the activation of NRF2 alleviates the occurrence and development of AD by enhancing the antioxidant defense, improving mitochondrial function, inhibiting neuroinflammation, and inhibiting ferroptosis (Qu et al., 2020)

after cells were cocultured with FOXO4DRI, the apoptotic and dead cells increase 8.9% (MLF), 5.5% (HLF)and 8.0% (MRC5) compare with TGF groups, suggesting a tendency of FOXO4 induced myofibroblast apoptosis