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These metabolic disturbances arise from disorders in iron absorption, transport, storage, and utilization, influenced by various external factors, diet, intestinal function, genetic predispositions, abnormalities in key proteins, changes in iron storage forms, and iron utilization disorders, all of which interact to contribute to PF ( 2+ , facilitates hydroxyl radical production via the Fenton reaction, exacerbating ROS-induced tissue damage, inflammation, and lipid peroxidation, thereby promoting fibrosis and lung function decline (Sun Y

Front Immunol 10:705

7, the K8A mutant protein showed significantly reduced DHAR activity