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Notably, similar inhibitions were observed both with the wild type enzyme and its cysteine mutants, suggesting that thiol conjugation is dispensable for GST inactivation by auronofin in contrast to other described targets 103

Furthermore, overexpression of GPX3 in adipocytes significantly reduced the expression of pro-inflammatory genes such as SAA3 , resistin and CCR2 induced by high glucose levels and attenuated hyperglycemia-induced insulin resistance [140]

doi:10.1016/j.envres.2013.07.005

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Unveiling the role of histone deacetylases in neurological diseases: focus on epilepsy

Using recommended doses minimizes risks