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So much of what we know right now comes from real-world patient experience rather than formal research data, he noted

Genotype should be considered when attempting to predict the potential CYP2C9 drug-drug interactions because of the possible genotype-dependent inhibition (22,23)

Consequently, we are unable to mechanistically validate the proposed signaling pathways or fully explain the patients unique PFS pattern and potential heterogeneity in treatment response

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The specific interaction of dinitrosyl-diglutathionyl-iron complex, a natural NO carrier, with the glutathione transferase superfamily: suggestion for an evolutionary pressure in the direction of the storage of nitric oxide

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