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Ferroptosis, first proposed by Stockwells group in 2012, is an iron-dependent modality of RCD and characterized by lipid peroxide accumulation, which leads to oxidative damage to cell membranes, and differs from other forms of RCD in several ways (Shojaie et al., 2020)

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Notably, these findings align with AKT1s well-established role in regulating glycolysis, glucose uptake, and TCA cycle flux via downstream effectors such as mTOR, GSK3, and FOXO1 [51,52,53]

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