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Notably, in this experimental model, the lower atherosclerosis in G6PD-deficient mice cannot be attributed to the reduced bioavailability of NO since apoE / eNOS double knock-out mice show increased atherosclerosis [68]

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Study of gastrointestinal tract viability and motility via modulation of serotonin in a zebrafish model by probiotics

Rodent physiology differs from human physiology in ways that are often relevant to drug development

Therefore, the increase in the trisulfide amount seemed to contribute to the strong antioxidant effect of the Si-based agent