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Conclusions and perspective The GSH system is one of the most significant members of the cellular antioxidant defensive system, which is essential for eliminating excess ROS and protecting the myocardium in the presence of pathological cardiovascular factors

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Oxidative stress mediates ethanol-induced skeletal muscle mitochondrial dysfunction and dysregulated protein synthesis and autophagy

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demonstrated that BAP1 can reduce the level of H2Aub1 in the SLC7A11 promoter region, increase lipid peroxidation, and induce ferroptosis [72]

Moreover, which immunological reactions or phases of the disease should be the target of ferroptosis