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A synthetic fragment related to thymosin beta-4 that acts as an extracellular repair signal and has accelerated re-epithelialisation and angiogenesis in dermal wound models

P = 0.001), and HDL-C ( F = 4.401

These findings support a model in which cold lethality of the slcr-46.1 mutant is not attributable to general sickness, but rather occurs in a copper-iondependent manner

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Safety and Limitations Topical use of GHK-Cu seems to be relatively safe