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Given the critical role of mitochondria in cellular metabolism and homeostasis, it is imperative to investigate how mitochondrial dysfunction induced by iron overload contributes to disease progression, as well as to explore mitochondrial-related pathways as potential therapeutic targets for various iron overload disorders
GSH is synthesiszed via two ATP-dependent processes that are catalysed by -Glu-Cys synthetase (GSH1) and GSH synthetase (GSH2), and it is mostly degraded by -Glutamyl transpeptidases (-GT), which split GSH into cysteinyl-glycine (Cys-Gly) and Glu residue
Patients who judge their progress entirely by the scan numbers at week 12 often conclude the treatment is not working
Check ClinicalTrials.gov for specific trial criteria
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