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This phenomenon has been linked to an increase in de novo lipogenesis (DNL), an elevated rate of free fatty acid (FFA) uptake, a decline in fatty acid oxidation (FAO), and an impaired hepatic excretion of FFA (Powell et al

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In a mouse model of triple-negative breast cancer, vitamin D supplementation reduced doxorubicin-induced cardiotoxicity by decreasing reactive oxygen species and mitochondrial damage, without compromising doxorubicin's anticancer efficacy [190]

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