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The dissociation/relaxation of nCRP into nnCRP occurs on necrotic, apoptotic, and ischemic cells, membranes of activated platelets, monocytes, and endothelial cells, and on the surface of microparticles, via phosphorylcholine binding and seems to be, as mCRP, proinflammatory (194196)

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doi: 10.1007/s11101-024-09991-3 149

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This powerful, multi-modal action is why the triple agonist is considered one of the most significant advances in metabolic medicine for both obesity and Type 2 Diabetes