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Urolithin A suppresses high glucose-induced neuronal Amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis

Fluorescence signals were visualized using a fluorescence microscope (Nikon Ts2R) and quantified by flow cytometry (NovoCyte 3000, Agilent, USA)

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Proc Natl Acad Sci United States America (2011) 108(38):160505

It has been shown to reset pathological gene expression to healthier states, which may counteract age-related dysregulation of biochemical pathways and support neuronal survival and function 11