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Animal models suggest an antifibrotic effect with delay in the endothelial-mesenchymal transition

This eliminates some estrogen-related side effects but introduces other issues that are often more severe

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This is especially crucial for patients whose metabolic flexibility has been compromised, as it helps the body efficiently utilize stored fats for long-lasting energy, rather than relying solely on rapid, easily exhausted, and lactate-producing glucose stores

Improvements appeared in lipid profiles, insulin sensitivity markers, and aminotransferase levels in subjects with elevated baselines

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