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When serine and threonine residues within the C truncation were replaced by alanine (Sec24C MUT), VRK2 could not increase phosphorylation of Sec24C MUT (Fig
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2019, Nov;1722:146331
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A rigorous exploration of the intricate signaling pathways involved, coupled with extensive verification across varied pathological frameworks, remains essential to fully characterize its pharmacological profile
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