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The unification hypothesis 226 suggests that several seemingly independent biochemical pathways that are overactivated in diabetes are actually caused by excessive intracellular glucose flux (Fig

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10.1016/j.ecoenv.2023.115194 51 SharmaS.SinghH.AhmadN.MishraP.TiwariA

As a result, RP-182 reprograms these M2-like TAMs into an antitumor M1-like phenotype, thereby augmenting both innate and adaptive antitumor immune responses