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This secretion is enhanced by increased Ca 2+ influx due to several PKA- and EPAC-dependent mechanisms: (1) the inhibition of ATP-regulated K + ion channels, (2) an increase in activity of L-type voltage-gated Ca 2+ channels (VGCCs), and (3) triggering the opening of non-specific cation channels [18,19,20,21]

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Keywords Alzheimer's disease, GHK-Cu, 5xFAD transgenic mouse, intranasal administration, amyloid plaques, neuroinflammation Introduction Alzheimer's disease (AD) is a complex disease that was the seventh leading cause of mortality in the US in 2022 [1]

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Electrolyte balance plays a role here as well

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