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These data show that where -BCR stimulation depends only partially on PI3K activation for the phosphorylation of AKT, gp34 1-179 depends exclusively on the Lyn-activated PI3K pathway for AKT activation

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Similarly, previous studies reported that circulating and liver FGF21 levels are upregulated, contributing to diabetes resistance, in systemic and liver-specific Gcgr -knockout mice [20, 25]