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The arginine-based ARG1/iNOS metabolic pathway regulates T cell activation thresholds via DC-T communication, thereby determining the balance of immune response and tolerance (157, 158)

Preclinical evidence in rats and mice points to the caudal brainstem and the mediobasal hypothalamus as sites that are necessary and sufficient for the ability of systemically administered GLP-1R agonists to reduce food intake, Rinaman said

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