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cAMP level can regulate the protein kinase A (PKA)-dependent activation of the cAMP response element-binding protein (CREB), which in turn upregulates the expression of PGC1 and inhibits LC3-II (Herzig and Shaw, 2018)

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(10) These proteins are often referred to as G-proteins, which, upon activation, might drive the generation of secondary messengers like cyclic adenosine monophosphate (cAMP) or inositol trisphosphate (IP3