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In the organoid models, mutated MEF2C led to altered microRNA levels, which contributed to the imbalance of inhibitory neurons and excessive glial proliferation

In our model, glucuronidation was also the main route of APAP elimination in most cases, and the range of contribution of each metabolic reaction was consistent with previous studies (Mcgill and Jaeschke, 2013) (Table 1)

Samples were analyzed using HPLC

229 Lactate, a byproduct of the glycolytic pathway produced by lactate dehydrogenases, has long been considered a waste product of glycolysis

With rising prevalence of both conditions globally, identifying effective pharmacological interventions remains a major clinical priority

Nat Rev Gastroenterol Hepatol 2015;12:231-242