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Additionally, its ability to improve insulin sensitivity and regulate adipose tissue inflammation makes it an attractive candidate for managing metabolic disorders beyond diabetes, such as non-alcoholic fatty liver disease (NAFLD) and metabolic syndrome

There is no autosomal dominant transmission of this syndrome because activating GNAS1 mutations are lethal when they occur early in embryogenesis [42]

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[5] [6] The GIP receptor component is thought to contribute to metabolic and appetite-related effects, whilst glucagon receptor activation may increase energy expenditure and promote fat breakdown (lipolysis) in the liver and adipose tissue

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