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For paracetamolcysteine, the cleavage of the carbonsulfur bond forms a product ion of m / z 182 (Fig

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Class II FINs induce ferroptosis by directly inactivating GPX4, such as RSL3 and ML162, which covalently bind to GPX4 to suppress its function, leading to an accumulation of toxic lipid peroxides and inducing ferroptosis [17, 105]

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