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Image Credit: Love Employee / Shutterstock In a recent study published in the Journal of Primary Care & Community Health , researchers detail a 10-month case report of a 34-year-old male with class 2 obesity and comorbid Alcohol Use Disorder (AUD)

In fact, by continuously depriving the liver of esterified fatty acids and inhibiting the hepatic glycolytic pathway, glucagon promotes the production of ketone bodies [41,42,43] Hepatic ketogenesis is boosted as a result, and mitochondrial fatty acid oxidation is improved [14, 43]

Researchers believe the bodys hunger and reward centers become more active again once the medication leaves the system

Nutritional support is essential: fiber intake should stay elevated (30+ grams daily), refined carbohydrates minimized, and meal composition optimized around protein and healthy fats