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The main mechanism responsible for Graves hyperthyroidism is the activation of the thyroid-stimulating hormone receptor (TSHR) by autoantibodies that act as agonists, causing thyrocyte proliferation and hyperfunction

The prognosis is difficult to predict, as few patients are known, but seems to vary significantly between different patients

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Among PDEs, PDE1A, PDE2A, and PDE10A have been identified as mRNA targets of FMRP (98)

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What patients typically report across a full cycle: Deeper and more restorative sleep: The most consistent early effect, often noticed within the first 3 to 5 nights of the cycle