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Furthermore, in vitro experiments in tumoral somatotrophs demonstrated that FLNA is required for SSTR2-induced reduction of cyclin D1 and caspase3/7 activation, suggesting a crucial role for FLNA in mediating antiproliferative and proapoptotic effects of SSTR2

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Depending on the intended site of action and nature of the gene-editing, the outcome of such treatment could differ drastically from the transient effects on both IgG and albumin achieved by first-generation FcRn antagonists

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