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Further, the appetite-suppressing effects of CCK are blocked with ablation of right vagal afferents, neurons in the lPBN and substantia nigra, and antagonism of D1R/D2Rs in the dorsal striatum [106], suggesting overlapping central mechanisms of CCK with RYGB

doi: 10.1055/s-2005-870424

Centrally [1], behavioral data provided anxiolytic [57,58,59], anticonvulsive [15,58,60,61,62,63,64], antidepressant effect [65,66,67], counteraction of catalepsy [68,69,70], and counteraction of positive and negative schizophrenia symptoms models [59,70]

Subsequently, these GABA-containing vesicles release GABA into the extracellular space through a Ca2+-dependent membrane fusion mechanism

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