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The proteomics and fibromyalgia: a perspective on the study of the inflammatory response in fibromyalgia

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In gene expression profiling, GHK-Cu treatment of human fibroblasts upregulated superoxide dismutase (SOD1, SOD2, SOD3) and glutathione-related genes while suppressing NF-kB-driven inflammatory signaling [3]

Since there are no direct clinical comparisons of the FDA-approved drugs, a systematic review was conducted to determine the similarities, advantages, and side effects of these medications

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