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(2023), who noted its ability to suppress inflammation, OS, and tubular cell apoptosis while promoting autophagy flux through a shift towards the intrarenal ACE2/Ang1-7 axis and away from the intrarenal ACE/Ang II axis

Defective mitochondrial DNA homeostasis in the substantia nigra in Parkinson disease

J Neuroimmune Pharmacol 14(4):608641 Cai A-J et al (2024) Recent advances and current status of gene therapy for epilepsy

p53 activation can suppress the mitochondrial tricarboxylic acid cycle and oxidative phosphorylation through inhibition of malic enzymes 1/2 or by inducing a PUMA-dependent metabolic switch (Jiang et al, 2013

For non-emergency concerns such as persistent injection site reactions or new neurological symptoms, contact your GP or NHS 111

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