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Cognitive functions

Future research should include comprehensive in vitro and in vivo studies to assess these compounds effects in a dynamic cellular environment and verify their selectivity for GSTP1 in cancerous versus normal cells

fergusonii , and E

Since previous studies have linked the activation of NRF2 with the repression of lipogenic gene expression 64 , we reasoned that GSH depletion causes oxidation and consequent inactivation of KEAP1 (a repressor of NRF2), thus leading to increased antioxidant and decreased lipogenic gene expression

Some neonatal studies show no depletion at therapeutic doses, suggesting babies may metabolize it differently

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