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That makes this conversation more nuanced

This is the same mechanism that drives semaglutide and the GLP-1 component of tirzepatide

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P=0.01), while improvement in fibrosis without worsening of MASH was reported in 45% of participants in the dapagliflozin group vs

Glucagon-like peptide-1 inhibits gastropancreatic function by inhibiting central parasympathetic outflow

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