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however, many clinical trials fail, and patients experience an unsatisfactory treatment response, likely owing to IC/BPS phenotype heterogeneity and the use of non-targeted interventions

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After pathogen (or danger) recognition, DCs undergo maturation and express on their membrane a panel of molecules that enable antigen presentation (MHC class II), T lymphocytes costimulation (i.e., CD40, CD80, CD86), and migration to lymph nodes (CXCR4, CCR7)

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