In summary, -HB has demonstrated clear potential in the prevention and treatment of T2DM and its complications and is expected to become a key focus in the fields of healthcare and pharmaceutical research in the future
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The NO synthase blocker L-NAME inhibited up-regulation of angiogenesis markers VEGF-A and CD34 by BPC 157, suggesting that BPC 157 promotes angiogenesis and then further repair the gastric mucosa through NO response
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This reveals distinctive NO-system clusters [3,11] that highlight distinct patterns of NO-related regulation and dysfunction [3,11] and distinctive NO-agents responses [3,11]
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