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Another proposed mechanism involves the inhibition of the transcriptional activity of nuclear factor erythroid 2-related factor 2, a key regulator of antioxidant defense systems, by elevated levels of homocysteine

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Thiol-rich antioxidants prevent FA cytotoxicity To address the causal contribution of FA-induced oxidative stress and GSH imbalance to cell death, we set out to test whether FA toxicity could be rescued by the antioxidants NAC, GSH-MEE, or Trolox (water-soluble vitamin E)