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This hypothesis aims to identify potential therapeutic targets for OCSCs

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10.2147/PROM.S155642 5

Substantial evidence indicates that mutations in several PD-associated genes (e.g., LRRK2, GBA, PINK1, PRKN, *DJ-1*) and the expression of risk alleles such as APOE4 exert their pathological effects by disrupting autophagy-related pathways, notably the autophagy-lysosomal pathway and mitophagy (3)

269 The benefits of TZDs in MASLD and fibrosis may be the increased adiponectin levels and the adiponectin-mediated effect on insulin sensitivity and hepatic fatty acid metabolism 270 and the downregulated fibrogenic factors and related pathways, inflammatory pathways like JAK/STAT and NF-B signaling pathways

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