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New York: Marcel Dekker, Inc

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Its mechanism of action involves stimulating both the glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) receptors

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Compared with a placebo or non-user comparator, this design minimizes immortal time bias and confounding by balancing disease severity or treatment eligibility in two groups, providing a more reliable real-world estimate of relative effects

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