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The key role of GSH as an antioxidant is demonstrated by many studies showing that experimental depletion of GSH levels which can be achieved with various chemicals the most used of which is buthionine sulfoximine (BSO) an inhibitor of GSH synthesis has a worsening effect in many disease models

Lowering BP to target cuts the rate of loss of eGFR in half, and adding a RAASi, as done in the Irbesartan Diabetic Nephropathy Trial, further reduced this rate considerably (Table 4)

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