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The primary, and perhaps most clinically significant, mechanism of action for BPC-157 revolves around the vigorous promotion of targeted angiogenesisthe complex physiological process through which new microvascular networks form from pre-existing blood vessels

Decreased lipidated ApoE-receptor interactions confer protection against pathogenicity of ApoE and its lipid cargoes in lysosomes

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Pre-stroke weight loss by glucagon-like peptide 1 receptor and neuropeptide Y receptor Y2 activation improves post-stroke functional recovery in male diabetic mouse models