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& Jarvi, K

(2018a) presented in vivo results on Scly expression and activity under the absence of Sec-rich Selenop, indicating the presence of other pathways of maintaining Sec supply for Scly, which remain to be identified

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conversely, normal SQOR activity eliminated excess sulfide and preserved CoQ 10 H 2 s antioxidant function, thereby mitigating cisplatin-induced oxidative damage and protecting the kidney (Cai et al., 2024)