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Previously, there is study revealed that promoting cellular intake of riboflavin by upregulating expression of SLC52A1 (a riboflavin transporter, also known as GPR172B/RFVT1) plays a crucial role in antisenescence via activating mitochondrial membrane potential and inhibiting the AMPK-p53 pathway [56]

The agency is also asking Lilly to collect data on thyroid cancer risk for at least 15 years

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