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Furthermore, PGC-1 suppresses the transport of NF-B p65 from the cytoplasm to the nucleus and the degradation of IB by regulating NF-B, thereby reducing A-induced neuronal death and inhibiting neuroinflammation, which in turn reduces mitochondrial damage and restores AD cognitive deficits (Zhang Y

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Our previous research indicated that overexpression of SREBP2 leads to increased expression of GLUT1/6 [6]

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