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It is also a cautionary illustration of how much context a mechanism carries: the best-characterized KPV effect depends on an intestinal transporter (PepT1) abundant in gut epithelium, which is one reason KPV has been explored mainly for oral delivery in bowel inflammation rather than as a general-purpose systemic anti-inflammatory

To further determine the role of YAP in SCD1 regulating the stemness of GCSCs, YAPsiRNA was transfected into HSC034 cells to suppress the YAP expression