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Apelin-13-induced mitochondrial iron excess is reversed by inhibiting SFXN1 and NCOA4 expression, which it also mitigates cardiac hypertrophy

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2012;62(4):696703 DOI: 10.1016/j.eururo.2012.03.051 2

Additionally, clinical samples should be employed to validate the correlation between serum CAT levels and markers of ferroptosis in articular cartilage (e.g., GPX4 activity) as well as disease severity in OA patients